Found 2 results
Filters: Keyword is Inclusion Bodies  [Clear All Filters]
2017
Dettmer U, Ramalingam N, von Saucken VE, Kim T-E, Newman AJ, Terry-Kantor E, Nuber S, Ericsson M, Fanning S, Bartels T et al..  2017.  Loss of native α-synuclein multimerization by strategically mutating its amphipathic helix causes abnormal vesicle interactions in neuronal cells.. Hum Mol Genet. 26(18):3466-3481.
2015
Dettmer U, Newman AJ, von Saucken VE, Bartels T, Selkoe D.  2015.  KTKEGV repeat motifs are key mediators of normal α-synuclein tetramerization: Their mutation causes excess monomers and neurotoxicity.. Proc Natl Acad Sci U S A. 112(31):9596-601.